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  <title>DSpace Community:</title>
  <link rel="alternate" href="http://dspace.bsuedu.ru/handle/123456789/52476" />
  <subtitle />
  <id>http://dspace.bsuedu.ru/handle/123456789/52476</id>
  <updated>2026-04-05T21:21:31Z</updated>
  <dc:date>2026-04-05T21:21:31Z</dc:date>
  <entry>
    <title>Neuroprotective properties of Na+/H+-exchanger isoform-1 inhibitor in experimental POAG</title>
    <link rel="alternate" href="http://dspace.bsuedu.ru/handle/123456789/62692" />
    <author>
      <name>Pobeda, A. S.</name>
    </author>
    <author>
      <name>Pokrovsky, V. M.</name>
    </author>
    <author>
      <name>Patrakhanov, E. A.</name>
    </author>
    <author>
      <name>Turpakova, A. V.</name>
    </author>
    <author>
      <name>Ustinova, A. I.</name>
    </author>
    <id>http://dspace.bsuedu.ru/handle/123456789/62692</id>
    <updated>2024-05-07T00:08:24Z</updated>
    <published>2023-01-01T00:00:00Z</published>
    <summary type="text">Title: Neuroprotective properties of Na+/H+-exchanger isoform-1 inhibitor in experimental POAG
Authors: Pobeda, A. S.; Pokrovsky, V. M.; Patrakhanov, E. A.; Turpakova, A. V.; Ustinova, A. I.
Abstract: Worldwide glaucoma is the leading cause of irreversible vision loss. The processes associated with the loss of retinal ganglion cells are multifactorial and have much in common with neurodegenerative diseases. Therefore the search for means to prevent the death of retinal neurons is an important task of modern pharmacology</summary>
    <dc:date>2023-01-01T00:00:00Z</dc:date>
  </entry>
  <entry>
    <title>Spontaneous remyelination following dimethyl sulfoxide-induced demyelination is accompanied by behavioral and neurological alteration in mice</title>
    <link rel="alternate" href="http://dspace.bsuedu.ru/handle/123456789/62691" />
    <author>
      <name>Kudryashov, N. V.</name>
    </author>
    <author>
      <name>Gorbunov, A. A.</name>
    </author>
    <author>
      <name>Sviridkina, N. B.</name>
    </author>
    <author>
      <name>Mironov, S. E.</name>
    </author>
    <author>
      <name>Tikhonov, D. A.</name>
    </author>
    <id>http://dspace.bsuedu.ru/handle/123456789/62691</id>
    <updated>2024-05-07T00:07:54Z</updated>
    <published>2023-01-01T00:00:00Z</published>
    <summary type="text">Title: Spontaneous remyelination following dimethyl sulfoxide-induced demyelination is accompanied by behavioral and neurological alteration in mice
Authors: Kudryashov, N. V.; Gorbunov, A. A.; Sviridkina, N. B.; Mironov, S. E.; Tikhonov, D. A.
Abstract: According to modern data acquired from in vitro experiments, Dimethyl sulfoxide (DMSO) is able to change the structure of myelin by decreasing synthesis of its main components and inhibiting oligodendrocyte genesis</summary>
    <dc:date>2023-01-01T00:00:00Z</dc:date>
  </entry>
  <entry>
    <title>Screening of anti-inflammatory activity of 4.5- dihydroisoxazol-5-carboxamide (PAR-2 inhibitors) based on formaldehyde oedema model among white lab rats</title>
    <link rel="alternate" href="http://dspace.bsuedu.ru/handle/123456789/62690" />
    <author>
      <name>Korsakov, M. K.</name>
    </author>
    <author>
      <name>Fedorov, V. N.</name>
    </author>
    <author>
      <name>Smirnov, N. A.</name>
    </author>
    <author>
      <name>Shetnev, A. A.</name>
    </author>
    <author>
      <name>Leonova, O. V.</name>
    </author>
    <id>http://dspace.bsuedu.ru/handle/123456789/62690</id>
    <updated>2024-05-07T00:08:07Z</updated>
    <published>2023-01-01T00:00:00Z</published>
    <summary type="text">Title: Screening of anti-inflammatory activity of 4.5- dihydroisoxazol-5-carboxamide (PAR-2 inhibitors) based on formaldehyde oedema model among white lab rats
Authors: Korsakov, M. K.; Fedorov, V. N.; Smirnov, N. A.; Shetnev, A. A.; Leonova, O. V.
Abstract: Rheumatoid arthritis (RА) is an immune-inflammatory disease intrinsic to up to 1.0% of the world’s population. Standard drugs for RA therapy are basic medications, glucocorticoids and non-steroid anti-inflammatory drugs, which often only ease or hinder the course of disease, not curing the patient completely</summary>
    <dc:date>2023-01-01T00:00:00Z</dc:date>
  </entry>
  <entry>
    <title>Features of bone remodeling and osteoreparation processes in modeling femoral fracture in genetically modified mice with impaired enzymatic regulation of steroid hormone metabolism</title>
    <link rel="alternate" href="http://dspace.bsuedu.ru/handle/123456789/62689" />
    <author>
      <name>Korokin, M. V.</name>
    </author>
    <author>
      <name>Gudyrev, O. S.</name>
    </author>
    <author>
      <name>Lebedev, P. R.</name>
    </author>
    <author>
      <name>Kochkarov, A. A.</name>
    </author>
    <author>
      <name>Pokrovskaya, T. G.</name>
    </author>
    <id>http://dspace.bsuedu.ru/handle/123456789/62689</id>
    <updated>2024-05-07T00:08:47Z</updated>
    <published>2023-01-01T00:00:00Z</published>
    <summary type="text">Title: Features of bone remodeling and osteoreparation processes in modeling femoral fracture in genetically modified mice with impaired enzymatic regulation of steroid hormone metabolism
Authors: Korokin, M. V.; Gudyrev, O. S.; Lebedev, P. R.; Kochkarov, A. A.; Pokrovskaya, T. G.
Abstract: The aim of this study was to evaluate the processes of bone remodeling and osteoreparation in modeling femoral fracture in mice with zero expression of 11β-HSD2 (11β-HSD2-/-) or both 11β-HSD2 and apolipoprotein e (11β-HSD2-/-/ApoE-/-)</summary>
    <dc:date>2023-01-01T00:00:00Z</dc:date>
  </entry>
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