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Please use this identifier to cite or link to this item: http://dspace.bsu.edu.ru/handle/123456789/52131
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dc.contributor.authorSoldatov, V. O.-
dc.contributor.authorKukharsky, M. S.-
dc.contributor.authorPuchenkova, O. A.-
dc.contributor.authorDeykin, A. V.-
dc.contributor.authorPokrovskiy, M. V.-
dc.date.accessioned2023-03-15T07:17:12Z-
dc.date.available2023-03-15T07:17:12Z-
dc.date.issued2021-
dc.identifier.citationRetinal abnormalities in transgenic mice overexpressing aberrant human FUS[1-359] gene / V.O. Soldatov [et al.] // Bulletin of RSMU. - 2021. - №4.-P. 10-15.ru
dc.identifier.urihttp://dspace.bsu.edu.ru/handle/123456789/52131-
dc.description.abstractThe aim of this work was to assess the structural and functional state of the retina in a murine model of ALS caused by overexpression of the aberrant FUS protein [1-359]. The retinal examination was carried out on 12 transgenic and 13 wild-type mice of 2.5-3 months of age. The study revealed not statistically significant higher level of ophthalmoscopic violations in FUS[1-359] miceru
dc.language.isoenru
dc.subjectmedicineru
dc.subjectmedical geneticsru
dc.subjectFUS proteinru
dc.subjecttransgenic miceru
dc.subjectALSru
dc.subjectretinopathyru
dc.subjectinflammationru
dc.titleRetinal abnormalities in transgenic mice overexpressing aberrant human FUS[1-359] generu
dc.typeArticleru
Appears in Collections:Статьи из периодических изданий и сборников (на иностранных языках) = Articles from periodicals and collections (in foreign languages)

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