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Please use this identifier to cite or link to this item: http://dspace.bsu.edu.ru/handle/123456789/64421
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dc.contributor.authorZhunusov, N. S.-
dc.date.accessioned2025-01-24T08:21:43Z-
dc.date.available2025-01-24T08:21:43Z-
dc.date.issued2024-
dc.identifier.citationZhunusov, N.S. Homozygous mice with mutant protein FUS[1-359] overexpression: Innovative possibilities for ALS treatment / N.S. Zhunusov // Research Results in Pharmacology. - 2024. - Vol.10, №4.-P. 137-141. - Doi: 10.18413/rrpharmacology.10.554.ru
dc.identifier.urihttp://dspace.bsu.edu.ru/handle/123456789/64421-
dc.description.abstractThis study investigates a mouse model with overexpression of the mutant FUS[1-359] protein, which can be used to evaluate the effectiveness of gene therapy and other pharmacological interventions for amyotrophic lateral sclerosis (ALS)ru
dc.language.isoenru
dc.subjectmedicineru
dc.subjectpharmacologyru
dc.subjectamyotrophic lateral sclerosisru
dc.subjectgene expressionru
dc.subjectgene therapyru
dc.subjectmouse modelru
dc.titleHomozygous mice with mutant protein FUS[1-359] overexpression: Innovative possibilities for ALS treatmentru
dc.typeArticleru
Appears in Collections:Vol. 10, № 4

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