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Please use this identifier to cite or link to this item: http://dspace.bsuedu.ru/handle/123456789/65887
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dc.contributor.authorKalitin, K. V.-
dc.contributor.authorMukha, O. Y.-
dc.contributor.authorVoynov, V. B.-
dc.contributor.authorSpasov, A. A.-
dc.date.accessioned2025-11-17T08:53:55Z-
dc.date.available2025-11-17T08:53:55Z-
dc.date.issued2025-
dc.identifier.citationThe prefrontal cortex as a target for the atypical antipsychotic RU-31 with 5-HT2A antagonistic activity in the treatment of cognitive symptoms / K.V. Kalitin, O.Y. Mukha, V.B. Voynov, A.A. Spasov // Research Results in Pharmacology. - 2025. - Vol.11, №3.-P. 98-108. - Doi: 10.18413/rrpharmacology.11.537. - Refer.: p. 106-108.ru
dc.identifier.urihttp://dspace.bsuedu.ru/handle/123456789/65887-
dc.description.abstractThis study establishes RU-31 as a promising therapeutic agent for the cognitive symptoms of schizophrenia, demonstrating that it reverses deficits in sensorimotor gating and normalizes PFC neuronal activity through selective 5-HT2A antagonismru
dc.language.isoenru
dc.subjectmedicineru
dc.subjectpharmacologyru
dc.subjectatypical antipsychoticsru
dc.subjectbenzimidazolesru
dc.subjectmolecular dockingru
dc.subjectprepulse inhibitionru
dc.subjectprefrontal cortexru
dc.subjectschizophreniaru
dc.subjectcognitive deficitsru
dc.titleThe prefrontal cortex as a target for the atypical antipsychotic RU-31 with 5-HT2A antagonistic activity in the treatment of cognitive symptomsru
dc.typeArticleru
Appears in Collections:Vol. 11, № 3

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