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Please use this identifier to cite or link to this item: http://dspace.bsuedu.ru/handle/123456789/66372
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dc.contributor.authorVoronkov, D. N.-
dc.contributor.authorStavrovskaya, A. V.-
dc.contributor.authorPavlova, A. K.-
dc.contributor.authorPotapov, I. A.-
dc.contributor.authorOlshansky, A. S.-
dc.date.accessioned2026-03-19T07:05:33Z-
dc.date.available2026-03-19T07:05:33Z-
dc.date.issued2025-
dc.identifier.citationSemaglutide reduces hypothalamic glial cell damage in a streptozocin-induced model of Alzheimer's disease / D.N. Voronkov, A.V. Stavrovskaya, A.K. Pavlova [et al.] // Research Results in Pharmacology. - 2025. - Vol.11, №4.-P. 66-77. - Doi: 10.18413/rrpharmacology.11.804. - Refer.: p. 75-77.ru
dc.identifier.urihttp://dspace.bsuedu.ru/handle/123456789/66372-
dc.description.abstractReactive changes in glial cells, as well as their dysfunction, particularly cerebrospinal fluid dynamics disturbances, are associated with multiple neurodegenerative diseases and Alzheimer’s disease (AD). Intraventricular administration of streptozotocin (STZ) is considered a model of sporadic AD, though data on hypothalamic glial cell changes, ependymal glia, and tanycytes in this model remain limitedru
dc.language.isoenru
dc.subjectmedicineru
dc.subjectpharmacologyru
dc.subjectastrocytesru
dc.subjectependymal cellsru
dc.subjectAlzheimer's diseaseru
dc.subjectstreptozotocinru
dc.subjectsemaglutideru
dc.titleSemaglutide reduces hypothalamic glial cell damage in a streptozocin-induced model of Alzheimer's diseaseru
dc.typeArticleru
Appears in Collections:Vol. 11, № 4

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